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Boyd Haley
I served as a medic in the U.S. Army from 1964 to 1966 and obtained my M.S. in Chemistry at the University of Idaho and my Ph.D. in Chemistry/Biochemistry at Washington State University in 1971. Additionally I was an NIH Postdoctoral Scholar in the Department of Physiology, Yale University Medical School from 1971 to 1974. From 1996 to 2005 I was Chair and Professor of Chemistry / Biochemistry in the Department of Chemistry at the University of Kentucky. Over the last two decades I have lectured throughout the world and testified before Congressional committees and the Institute of Medicine regarding various aspects of mercury toxicity and neurological diseases.
Boyd Haley PhD explains why the apolipoprotein-4 (APOE-4) genotype represents a genetic susceptibility to mercury toxicity as a pathogenetic factor and a moderator of Alzheimer's Disease.
This article presents excerpts from a letter Boyd Haley PhD wrote to congressman Dan Burton as a rebuttal to a May 11th, 2001 letter by Robert M. Anderton, D.D.S., and President of the American Dental Association (ADA), challenging Boyd's statements to the Committee on Government Reform. Additionally, Boyd gives a complete dissection of the many flaws, including misleading data manipulation in the Saxe Alzheimer's study.
OSR (Oxidative Stress Relief) developed by Boyd Haley is a powerful antioxidant. High levels of free radicals and oxidation can lead to oxidative stress. The body fights oxidation by producing glutathione. Mercury inhibits the production of glutathione, which then may not be able to keep up with the production of free radicals. OSR assists glutathione with scavenging free radicals.
In the past 20 years I have concentrated my research on the effects of mercury toxicity on human health. Specifically, I have researched and evaluated the contributions of dental amalgam, biologics and vaccines on the human body burden of mercury and organic-mercury compounds and the potential effects of these compounds on specific enzymes and cells.
Emeritus Professor of Chemistry, Boyd Haley, Ph.D. from the University of Kentucky outlines the flaws in two studies that were conducted in Portugal and New England that compared youngsters with dental mercury fillings with those who were amalgam free. In designing their studies, the authors of these two papers evidently ignored recent research findings about mercury toxicity, particularly the results strongly suggesting that the level of mercury in blood, urine or feces may be influenced more by the child’s ability to excrete mercury than by his or her total mercury exposure.
Mercury (as Hg2+) exposure to neurons in culture has been shown to produce three of the widely accepted pathological diagnostic hallmarks of AD. These are elevated amyloid protein, hyper-phosphorylation of Tau, and formation of neurofibillary tangles. The hypothesis is that mercury and other blood-brain permeable toxicants that have enhanced specificity for thiol-sensitive enzymes are the etiological source of AD
Boyd Haley PhD explains the benefits of Oxidative Stress Relief (OSR) and the limitations of chelators DMPS, DMSA & EDTA.
In this video Boyd Haley PhD discusses Urinary Porphyrn Profile Testing for Mercury Toxicity.
Boyd Haley P.h.D. discusses the importance of testing Glutathione to determine mercury toxicity.
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